Keratolytic Effects
By Cosmetic Peat Editorial Team Updated June 2026
Skin constantly sheds its outer layer. Normally this happens invisibly — dead cells detach one by one and wash away. But in conditions like psoriasis, dandruff, and seborrheic dermatitis, the process goes wrong. Cells pile up faster than they shed, forming thick plaques, flakes, or scales. Keratolytic agents break these accumulations down — and peat does it through three compounds working together.
How it works
Think of dead skin cells as tiles held together by mortar. To remove them, you need to dissolve the mortar. Peat attacks these bonds from three angles simultaneously:
Humic acids soften keratin protein through direct interaction with its structure — loosening the “mortar” between dead cells. This is a gentle, gradual action rather than the aggressive stripping of a chemical peel.
Organic acids in peat work like a mild version of the alpha-hydroxy acids (AHAs) used in cosmetic exfoliants. They weaken the bonds between corneocytes — the flat, dead cells of the outermost skin layer — allowing them to detach more easily.
Sulfur compounds break the disulfide bonds that cross-link keratin proteins. This is the same mechanism behind sulfur-based dandruff shampoos and psoriasis treatments — peat delivers it alongside anti-inflammatory and antimicrobial compounds rather than in isolation.
The result is gentle but effective exfoliation. A 12-week study using peat extract showed visible reduction in scalp squames (flakes) under microphotography (Bovcon 2012). Clinical observations from psoriasis patients report plaque softening and reduction during peat treatment courses (Leibaschoff 2005).
Why it matters
Keratolytic action is especially important because dead skin buildup is both a symptom and a barrier to treatment. A thick psoriatic plaque blocks topical anti-inflammatory compounds from reaching the living skin beneath it. By softening and removing the buildup, peat’s keratolytic mechanism clears the way for its own anti-inflammatory and moisturizing compounds to work on the underlying problem.
How It Works
| Targets | Keratin protein in stratum corneum |
| Pathway | Softening and dissolution of intercellular bonds in dead skin layer |